Kelly Gibas

Job Titles

Highlight

Professor Gibas' research includes the exploration of functional integrative treatments for metabolic pathologies, insulin resistance, diabetes and neurodegeneration, with a focus on the role of the CNS to modulate systemic fuel flux/nutrient partitioning under deranged insulin signaling.

Started at Bethel

2016

Education

  • Bethel College - B.A. in Communications, 1991
  • Bethel University - M.A. in Counseling Psychology, 2014
  • Arizona State University - Ph.D. in Behavioral Health , 2017
  • Southern California University - Functional Medicine Practitioner (CFMP), 2018

Courses Taught

August 2016 - Present

HAS445 Advanced Lab Practices

HAS379 Integrative Human Physiology

HAS399 Physiological Assessment

HAS398 Physiological Assessment Lab

HAS370 Functional Human Nutrition

BIOL610 Clinical Physiology - Bethel University Physician Assistant Program

Publications

M.K Gibas, K.J. Gibas, Induced and controlled dietary ketosis as a regulator of obesity and metabolic syndrome pathologies, Diab Met Syndr: Clin Res Rev (2017), http://dx.doi.org/10.1016/j.dsx.2017.03.022 (RCT, March 2017) PMID: 28433617 https://clinicaltrials.gov/ct2/show/NCT03047447

Gibas, K.J (2017). The Starving Brain: Overfed meets undernourished in the pathology of mild cognitive impairment (MCI) and Alzheimer's disease (AD). Neurochemistry International,https://doi.org/10.1016/j.neuint.2017.09.004. PMID: 28899812

Gibas, K.J. The Retrograde Signal: Glucose dependency marks the cancerous phenotype. Austin Diabetes Research, Austin Diabetes Res. 2017; 1(1): 1065.

Gibas K.J. The Retrograde Signal: Dependency on Glucose Marks the Cancerous Phenotype. Austin J Nutr Metab. 2017; 4(2): 1051.

Gibas K. Commentary: The Starving Brain: Overfed meets undernourished in the pathology of mild cognitive impairment (MCI) and Alzheimer's disease (AD). J Immunological Sci.(2018); 2(2): 1-3.

Dahlgren K, Gibas KJ. Ketogenic diet, high intensity interval training (HIIT) and memory training in the treatment of mild cognitive impairment: A case study. Diabetes & Metabolic Syndrome: Clinical Research & Reviews. 2018. doi:10.1016/j.dsx.2018.04.031. PMID: 29678606

Brown D, Gibas KJ. Metabolic syndrome marks early risk for cognitive decline with APOE4 gene variation: A case study. Diabetes & Metabolic Syndrome: Clinical Research & Reviews. 2018. doi:10.1016/j.dsx.2018.04.030. PMID: 29706312

Gibas KJ, Halikas A. AMPK induced memory improvements in the diabetic population: A Case Study. Diabetes & Metabolic Syndrome: Clinical Research & Reviews. 2018. doi:10.1016/j.dsx.2018.04.033. PMID: 29748034

Morrill SJ, Gibas KJ. Ketogenic diet rescues cognition in ApoE4 patient with mild Alzheimers disease: A case study. Diabetes & Metabolic Syndrome: Clinical Research & Reviews. 2019;13(2):1187-1191. doi:10.1016/j.dsx.2019.01.035. PMID: 31336463

Cox N, Gibas S, Salisbury M, Gomer J, Gibas K. Ketogenic diets may reverse Type II diabetes and ameliorate clinical depression: A case study. Diabetes & Metabolic Syndrome: Clinical Research & Reviews. 2019. doi:10.1016/j.dsx.2019.01.055. PMID:31336509

Stoykovich S, Gibas K. APOE e4, the door to insulin-resistant dyslipidemia and brain fog? A case study. Alzheimers & Dementia: Diagnosis, Assessment & Disease Monitoring. 2019;11:264-269. doi:10.1016/j.dadm.2019.01.009. PMID: 30923733

Gibas KJ.Dietary Ketosis a Metabolic Sister to Calorie Restriction (CR): Fatty acids activate AMPK energy circuits modulating global methylation via the SAM/SAH axis. Randomized clinical trial (2019): NCT0331917 https://clinicaltrials.gov/ct2/show/NCT03319173

Gibas, KJ. Photobiomodulation & ketogenic diet for treatment of mid-periphery retinal disorders (diabetic retinopathy, dry AMD and hard drusen formation) for Alzheimer's disease prevention. Randomized clinical trial (2019): NCT03859245. https://www.clinicaltrials.gov/ct2/show/NCT03859245

Presentations

Gibas, K. (November 2017). Dietary Ketosis a Metabolic Sister to Calorie Restriction (CR): Fatty acids activate AMPK energy circuits modulating global methylation via the SAM/SAH axis in mild cognitive impairment (MCI).Poster presentation at Cell Symposia: Big Ideas in Neuroscience Conference, 2017, Arlington, VA. http://www.cell-symposia.com/big-questions-in-neuroscience-2017/conference-program.asp

Gibas, K. (December 2018). Mouth breather? Enduring patterns of hypocapnia induce cerebral hypoxia and consequential cognitive impairments mediated by aberrant HIF-1 survival signals common to the cell danger response.Poster presentation at Cell Symposia: Metabolites as Signaling Molecules Conference, 2018, Seattle, WA. http://www.cell-symposia.com/metabolites-2018/conference-program.asp

Gibas, K. & Stoykovich, S. (April, 2019). ApoE4, the door to insulin-resistant dyslipidemia and brain fog? A case study. Poster presentation at American Diabetes Association/WCITD - 2019 New York, NY. https://cmoffice.kenes.com/cmsearchableprogrammeV15/conferencemanager/programme/personid/anonymous/WCITD19/normal/b833d15f547f3cf698a5e922754684fa334885ed#!abstractdetails/0000394330

Certificates and Licenses

Licensed Professional Clinical Counselor, LPCC

Certified Functional Medicine Practitioner, CFMP

Professional Organizations, Committees, and Boards

Societies/Scholarships: Phi Kappa Phi Honor Society, Arizona State University academic scholarships, 2015, 2016, & 2017

 

Hobbies and Interests

Dr. Gibas enjoys reading, running, & spending time with family and friends.

Research interests

Professor Gibas' research includes the exploration of functional integrative treatments for metabolic pathologies, insulin resistance, diabetes and neurodegeneration, with a focus on the role of the CNS to modulate systemic fuel flux/nutrient partitioning under deranged insulin signaling.

Website

https://www.linkedin.com/in/kelly-gibas-582b7462/; www.bristleconemedical.com